Answer :
Answer:
The options
A)hippocampus
B)basal ganglia
C)hypothalamus
D)cerebellum
The CORRECT ANSWER IS D)
D)cerebellum
Explanation:
Apart from the coordinating process, the cerebellum is involved in motor learning, emotional behavior, and fear memory.
Fear learning is widely shown in the variation of autonomic and somatic responses, as in heart rate and freezing, promoted by a neutral stimulus which could have been formally grouped with a painful one.
Variation or changes of the vermis influences these responses, and its reversible inactivation in the period of consolidation which is seen to impair fear memory.
The neural correlate of cerebellar activities in fear consolidation is shown by a behaviorally promoted long-term rise of synaptic effectiveness and efficiencies among parallel fibers and a Purkinje cell.
Likely synaptic variations after fear conditioning are well fully observed in the amygdala and hippocampus, suggesting an interplay among emotional circumstances of scenario and the different variations in neural process.
Furthermore, in hotfoot mice, that shows a basic lack of parallel fiber to Purkinje cell synapse, short- and long-term fear memories are influenced.
All these data gives a foundational background on the concept that the cerebellum is majorly involved in fear learning.
The functional interplay of the vermis with hypothalamus, amygdala, and hippocampus do infer to a more complex activities of the cerebellum as a form of an united network that basically influences emotional behavior.
Answer:
Cerebellum
Explanation:
In addition to coordinating movement, the cerebellum participates in
motor learning, emotional behavior, and fear memory. Fear learning
is reflected in a change of autonomic and somatic responses, such
as heart rate and freezing, elicited by a neutral stimulus that has been
previously paired with a
painful one. Manipulation of
the vermis affects these
responses, and its reversible
inactivation during the
consolidation period impairs
fear memory. The neural
correlate of cerebellar
involvement in fear
consolidation is provided by a
behaviorally induced long-
term increase of synaptic
efficacy between parallel fibers
and a Purkinje cell. Similar
synaptic changes after fear
conditioning are well
documented in the amygdala
and hippocampus, providing a
link between emotional
experiences and changes in
neural activity. In addition, in
hotfoot mice, with a primary
deficiency of parallel fiber to
Purkinje cell synapse, short-
and long-term fear memories
are affected. All these data
support the idea that the
cerebellum participates in fear
learning. The functional
interconnection of the vermis
with hypothalamus, amygdala,
and hippocampus suggests a
more complex role of the
cerebellum as part of an
integrated network regulating
emotional behavior.